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Butyrate inhibits inflammatory responses through NFkappaB inhibition: implications for Crohns disease

Under normal physiological conditions the NO increase might be controlled by S-nitrosylation/glutathionylation of eNOS as an immediate response or by similar modification/activation of SERCA in steady-state regulation

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( A ) ATP generation and ( B ) intracellular cAMP accumulation were analyzed as detailed in the Supplemental Methods from WT sAC +/+ caSMCs incubated for 10 minutes with 0.1 nM to 1 M GLP-1(2836) or scrambled control

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